i-blocki (Tropix Inc)
90
Structured Review
Tropix Inc
i-blocki
I Blocki, supplied by Tropix Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/i-blocki/i+blocki/pm16005083-85-13-14
Average 90 stars, based on 1 article reviews
I Blocki, supplied by Tropix Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/i-blocki/i+blocki/pm16005083-85-13-14
Average 90 stars, based on 1 article reviews
i-blocki - by Bioz Stars,
2026-10
90/100 stars
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Incubation:Article Title: Tumor necrosis factor-alpha mediates the proliferation of rat C6 glioma cells via beta-adrenergic receptors. Article Snippet: In the present study, we observed that isoproterenol, a h-adrenergic receptor (h-AR) agonist, stimulated rat C6 glioma cell proliferation, while propranolol, a h-AR blocker, greatly reduced the proliferative effect of TNF-a on C6 cells.. The gene and protein expressions of both h1and h2-ARs were enhanced in C6 cells after TNF-a treatment, and the increase in h-AR was due to an increased number of binding sites and not due to increase in receptor affinity.. We further showed that protein kinase C (PKC) was involved in the TNF-a-induced h-AR expression. Blocking Assay:Article Title: Tumor necrosis factor-alpha mediates the proliferation of rat C6 glioma cells via beta-adrenergic receptors. Article Snippet: In the present study, we observed that isoproterenol, a h-adrenergic receptor (h-AR) agonist, stimulated rat C6 glioma cell proliferation, while propranolol, a h-AR blocker, greatly reduced the proliferative effect of TNF-a on C6 cells.. The gene and protein expressions of both h1and h2-ARs were enhanced in C6 cells after TNF-a treatment, and the increase in h-AR was due to an increased number of binding sites and not due to increase in receptor affinity.. We further showed that protein kinase C (PKC) was involved in the TNF-a-induced h-AR expression. |